Case 1- A 38-year-old female
presented with initial complaints of
headaches, neck, midback, and low back pain
resulting from a roll-over motor vehicle
accident in January 1988.
She sustained a
compression fracture of the L1 vertebral
body, with findings of small disc bulges in
the cervical and lumbar spine and a reversal
of her cervical curve. She was initially
hospitalized for 1 week and continued her
treatment with medications, physical
therapy, an chiropractic manipulative
therapy for approximately 1 year. In 1992,
another patient advised her of the procedure
of manipulation under anesthesia, at which
time she sought treatment at the office. The
treatment had not worked since the accident,
was on total disability, and was using
alcohol for pain relief. Examination prior
to MUA included a pain level of 9 (on a 0-10
scale) with greatest pain being in the
cervical spine (leading to headaches) and in
the area of L1 compression fracture.
Straight leg raise (SLR) caused pain in her
low back bilaterally and down her right leg
at 30 , and at 45 on the left side.
Bragard's sign was positive on the right.
Cervical and lumbar motions were decreased
at least 50% in all directions and 70% in
flexion with pain on all movements. Her
paraspinal muscles were light and tender to
touch from T8 to S1. She had referred pain
down both arms and legs, worse on the right.
She underwent 3 consecutive days of MUA in
May 1992. to the cervical, thoracic, and
lumbar spine and also received physical
therapy three times per week in the form or
interferential electrical stimulation,
ultrasound, heat, and massage. She reported
great relief from the pain at the end of the
three trials of MUA. One month post-MUA her
pain level (0-10) was 1 and her headaches
had resolved. SLR was 85 bilaterally without
referred symptoms. Lumbar range of motion
(ROM) was within 90% of normal and cervical
ROM was normal. She claimed no referred pain
and had normal sensation. Six months
post-MUA, SLR was 80 . Pain and stiffness
had slightly increased to2/20 in the
cervical and a 3/10 in the lumbar spine
region. This was significant improvement in
her pain and functional level from her-MUA
procedure condition. At 1 year
post-procedure she reported that pain
reached a maximum of 4/10 in the region of
the L1 compression fracture when doing
activities of bending, lifting, and
twisting.
REFERENCES (JNMS:
Journal of the Neuromusculoskeletal System
4:102-105, 1996)